Structure/Class |
- These antibiotics contain a macrocyclic lactone ring, e.g. erythromycin, azithromycin and clarithromycin.
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Pharmacodynamics |
- Functions by inhibition of protein synthesis by binding to the 50S ribosomal RNA
- Spectrum of activity is against Gram+ve, Gram-ve and intracellular organisms.
- Gram+ve: staph, strep, pneumococci and cornebacterium
- Gram-ve: Neisseria, bordatella, rickettsia and campylobacter
- Intracellular: Mycoplasma, chlamydia, listeria, legionella and certain mycobacterium.
- As such, clinical indications as follows:
- Respiratory infections
- Community acquired pneumonia
- Chlamydial infections
- Note that these macrolides may be both bacteriostatic and bactericidal, depending on concentration of drug and organism susceptibility.
- Resistance is generally plasmid encoded. Important to remember that cross resistance is complete. The following patterns of resistance are identified:
- Reduced entry into cell
- Increased efflux of drug
- Modification of binding site at ribosome
- Production of esterases that hydrolyze macrolides.
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Absorption/administration |
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Distribution |
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Metabolism |
- Metabolised in liver to inactive products.
- It is a strong inhibitor of CYP450 enzymes and therefore increases the levels of drugs like warfarin and theophylline.
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Excretion |
- Mainly in faeces and in bile. Minimal in urine. As such, there is no need for dose adjustment in renal failure.
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Interactions |
- It is a strong inhibitor of the CYP45- system, and therefore increases levels of drugs such as warfarin, phenytoin and theophylline.
- It also increases the serum concentration of digoxin by increasing its oral bioavailability.
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Adverse events |
- GIT side effects are common (remember that erythromycin is used as a prokinetic in the ICU setting) – hence, nausea, vomiting and diarrhea are common.
- Acute cholestatic hepatitis (hypersensitivity reaction). Characterized by fever, jaundice and impaired LFTs.
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Dosing/administration |
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Toxicology |
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Withdrawal syndrome |
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Special notes |
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